Sintaxis de muestra

"It is often that in the building of perfect beasts that the issues of health and longevity become a primary concern. Oddly enough many, not of my design, opt to introduce health concerning factors at a phenomenal rate as a result of misinformation or critics. Misinformation in itself appears to be a commodity at times. Perhaps this is why I am criticized by some for my distinct lack of long term oral anabolic/androgenic steroids utilized in cycle outline discussions. For the critics I say: "Like I care what you think."

Oral AAS did posses a few distinct advantages and did have some risk-to-effect value when utilized with responsible common sense. As you know form the first "Chemical Muscle", most oral steroids were usually noted to be very liver toxic due to their c17-alkylated structures. (Primobolan Acetate tabs was not c17-alkylated) Remember; If a beast had screwed up their liver, serious bodybuilding (and health) would have been pretty much history.

The good effects of orals were elevated liver release of IGF-1 due to a liver detoxification (and deactivation) Action/Reaction, no extra injections, and convenience. When used for brief periods of 3-6 weeks liver enzyme values usually returned to normal within a 4-8 week period after discontinuance. But for the wiser athlete limited use was best. Let's briefly look at some common oral AAS and the possibilities we had employed, again. Each has been successfully layered into various phases.

ANADROL-50 (Oxymetholone) Anadrol was probably the all time favorite mass

and strength building oral AAS. At dosages of 100-250 mg/d, many realized weight gains of 1 5-25 lbs...n only a few weeks. Users raved about unbelievable strength and recovery as well. Unfortunately, the majority of weight gain was water retention and to a much lesser extent increased red blood cell count. Post-cycle lean mass retention sucked unless a high anabolic AAS such as Nandrolone, Equipoise, Winstrol-Depot, or Primobolan Depot were stacked with it, or in my experience more wisely utilized in a Max Androgen Phase. (And Anadrol was discontinued at least 10-14 days before the high anabolic of course)

Anadrol was commonly used the last few weeks pre-contest also. This was done to increase training intensity, post work-out recovery, increase vascularity, and induce a distinct fullness/hardening effect. When used pre-contest, Anadrol required the use of Nolvadex or other estrogen receptor antagonist.

Even with the use of a finasteride-type drug, Anadrol tended to cause balding for some. It also gave many athletes raging gyno in the upper dosage ranges. Anadrol does not actually aromatize but does posses progesterone like qualities.

Progesterone is an estrogen that causes gyno as well as serious water retention. Faslodex (fulvestrant) has been the better estrogen antagonist during administration of any AAS with progestin activity. Why, as I said prior, the drug works by down- regulating estrogen and progesterone receptor counts. Less receptors means less activity.

There was a debate as to whether or not Anadrol did, or could convert to, a DHT or DHT derivative. I now feel both sides are unlikely true or right. Anadrol was such a potent androgen that it attached itself to sex tissue specific androgen receptors in the scalp and prostatic areas. Finasteride did seem to posses some receptor-site blocking qualities and therefore was helpful.

Interestingly, Anadrol did not induce strong activation of androgen receptor- sites in muscle tissue. But it did cause impressive muscle mass gains. Ironic, but obviously Anadrol worked.

Sadly, Anadrol was seriously liver toxic. Only Methyltestosterone rivaled it for toxicity and Halotestin beat it for liver destruction.

Due to serious water retention during Anadrol use pre-contest, a diuretic would be necessary the last 2-3 days pre-contest. Remember Anadrol causes serious HPTA suppression.

ANAVAR (Oxandrolone) Anavar was actually a very potent AAS. I can almost

here you laughing, but milligram for milligram, Anavar caused a greater anabolic response than Anadrol. Oddly enough, even though Anavar was a c17-alkylated structure and elevated liver values, it did not seem to aggravate liver problems. Anavar did not convert to DHT, nor did it aromatize to estrogen.

Pre-contest, Anavar significantly increased hardness and aided in lean mass retention during calorie-restricted periods. During use, some strength gains were realized but weight gain was slow even with a higher calorie intake. However, the weight was pure lean muscle and was mostly retained post-cycle even without HPTA stimulation. This was due to Anavar's distinct lack of HPTA suppression qualities and low androgenic value.

An interesting use for Anavar and other non-HPTA suppressing AAS was lean mass retention after a heavy AAS cycle. This was very effective while HPTA stimulators kicked in endogenous androgen production and catabolic hormone levels normalized.

Elevated ATP levels significantly aided in post-cycle lean mass retention. And guess what? Anavar increased creatine phosphate synthesis. I know of several fitness competitors who stacked 25 mg of Anavar daily with 20-30 mg of Nolvadex and either 80-120 mcg of Clenbuterol and/or an ephedrine /caffeine stack, daily. They got lean, hard, and often didn't need thyroid drugs if a good diet plan was followed.

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Several hard-core male bodybuilders commonly used 40-80 mg/d of Anavar during the last 3-4 weeks of contest prep to increase hardness and preserve lean body mass. Like I said, "interesting". The down side was expense. It should seem apparent that Anavar has been successfully utilized by Frank N. Steroid as a replacement for non-HPTA suppressing AAS during Low Dose AAS Phases. The dosage equaled 0.25 mg per Ib/d.

There was a black market version of Oxandrolone circulating that allowed for injection type administration. The problem was that not all AAS are effective to a great degree when the liver deactivation factor is removed to a significant degree...and this was one.

DIANABOL (Methandrostenolone) Dianabol has had a long history in sports of

all types, like since the '50's, in fact. In most cases, 50 mg/d of Dianabol caused the same results as 1 50-200 mg/d of Anadrol-50 and was somewhat easier on the liver.

Usually a dosage of 5mg/25 Ib of bodyweight daily promoted excellent strength and mass weight gains for any hard-core bodybuilder. Personally, I just did not believe a dosage above this was more effective or had risk-to-effect value. (If the Dianabol utilized was real and dosage per tablet/liquid in gel caps was accurate.)

I felt Dianabol was excellent at aiding in a faster androgen plasma level during the first 7-10 days of a Max Androgen Phase. And most reported a sensation of feeling "Great" during employment.

Dianabol did aromatize significantly, and did posses some DHT receptor stimulatory qualities. In this case, Finasteride did allow for some receptor antagonist value and was fairly effective for inhibition of DHT formation.

Aromatization was usually handled with most aromatization inhibitors such as Cyclophenil, Arimidex, Proviron, or Teslac. However, a greater benefit was realized with Nolvadex. This was due to Nolvadex being an estrogen antagonist. Since antagonist only "block" estrogen receptor-sites, the elevated circulatory estrogen levels caused IGF-1 production increases. Which was cool if there was a sufficient androgen level, of course!

However, when Dianabol was layered into an AAS cycle or utilized alone for more than 21 days, an aromatase inhibitor needed to be added at least 7 days before discontinuance of all AAS. If not, post-cycle estrogen levels became a problem.

It should be realized that Dianabol was most potent in its effects as an oral, not injection form AAS. This was due to remarkable IGF-1 release from the liver during detoxification of its molecule, which was of course a c17-alkylated structure. Unfortunately, Dianabol also had serious HPTA suppressing effects.

Since Dianabol caused high water retention, it was not normally utilized as a contest prep drug. For mass cycles with good post-cycle lean mass retention, Dianabol was commonly stacked with, or in the case of my beast, phased with a high anabolic drug such as Nandrolone, Equipoise, or Primobolan Depot. These high anabolic drugs had a respectable affinity for androgen receptor-sites and solidified Dianabol induced mass into high quality lean muscle tissue...when the protocol was properly structured.

A plus for Dianabol was its distinct cortisol suppression qualities. In fact, 50- 70% of cortisol's effects were blocked at a dosage of 5mg/25 Ib daily. About 10 mg daily was noted in available literature as equal in activity to an average male's endogenous androgen production.

Editor's Note: At the time of this writing, the research team has discovered an oral

"pro-Dianabol" compound that HM Gears should be making available within the next few months. It works EXACTLY like Dianabol, but you'll just need to take more

milligram wise. Cool part is that it'll be fairly cheap, about $40 or so for a month's supply (which is the equivalent of a 15-20 mg daily dosage of REAL dianabol!. We are finalizing the patent on this compound and trying to get a good price for our first initial batch run (first runs always end up costing more due to lack of volume, setup costs, etc.). By the lime this book is published and you are reading this, it will hopefully be available for sale at www, hmgear. Com

HALOTESTIN (Fluoxymesterone) Personally, I hated the method of use most

employed with this drug. Though its high androgenic characteristic increased hardness pre-contest, and fueled serious training intensity, it ate livers for breakfast.

Halotestin did little at the androgen receptor-sites in muscle tissue. However, it did do serious activation at steroid receptor sites in the brain in comparison. The result was hard-core intensity and a bad attitude problem for most athletes who were susceptible.

As I mentioned prior, I did not think of the normal 8-12 week progressive dosage administration schedule as "best utilization". Later I will explain the approach my beast used to decrease liver stress. I have noted that Halotestin 10mg 2xd is a common prescribed androgen therapy as I write this story.

Halotestin is a DHT derivative. Blood test results confirmed a significant elevation in DHT during administration. But stock in Proscar probably went up! Gyno was not a problem with Halotestin due to a lack of aromatization. In fact, some individuals have reduced their existing gyno due to the use of this drug. It also did not cause water retention, another pre-contest plus.

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maintenance during calorie restriction was a plus. I could not find a main stay use for it except possibly during the last 3-4 weeks of contest prep... and then, only as a last choice of a high androgen.

METHYLTESTOSTERONE Don't look for any good words for this liver killer,

gyno causing, headache inducing drug from me. Funny irony to think about. Last year while researching a possible pro hormone structure, I discovered a way to introduce 4- Androstenediol into the system in a way that induced GI track conversion to Methyltestosterone and absorption at a very high rate. (And it was legal)

WINSTROL Orals (Stanozolol) I believe milligram for milligram Winstrol was

the most effective oral. I have written and said it several times; when compared to Anadrol, Dianabol, or Anavar, Winstrol kicked butt. Unfortunately it was an alkyl 17 oral.

Winstrol was not a "great" strength drug but lean muscle tissue growth was far superior. Winstrol seriously stimulated androgen receptors and aided in hardness to some extent due to low water retention. Obviously, Winstrol increased protein synthesis on two levels. First as an anabolic steroid and second as an oral IGF-1 stimulator.

Winstrol did not convert to DHT or aromatize to estrogen. However, it did cause some hair loss. Since it has been noted to attach easily to scalp androgen receptor sites this was not a major surprise. It was much milder on the liver than Dianabol or Anadrol. And certainly was more liver friendly than Halotestin. But then, so is an ice pick.

Another plus for Winstrol was its structural similarity to progesterone. This means it was noted to bind with progesterone receptors and yet inhibit progesterone's effects. This was obviously like Nolvadex in that it blocked receptor sites. For this reason it has been successfully been utilized as part of an oral stack (When orals became necessary) with either Dianabol or Anadrol. As a mass cycle the protocol required using 25-50 mg/d of Winstrol and 5-10 mg/50 Ib bodyweight daily of Dianabol or 100-200 mg/d of Anadrol. 50-150 mg/d of Anadrol for pre-contest phases during the last 3-4 weeks was fairly effective as well.

Winstrol orals or injectable stacked well with Anavar for brief periods following longer AAS protocols and during initial HPTA restimulation. Winstrol had little effect upon HPTA function. So this had some real benefit in aiding post-cycle lean mass retention. Winstrol was regularly stacked with most other AAS as a synergistic component of any protocol by a few successful beasts.

Oral Winstrol tabs and Winstrol Depot /V were actually identical. Orals were quite expensive at 25 mg/d or even 50 mg/d. As I explained in the first "Chemical

Muscle", simply orally ingesting the injectable version (1/2 m = 25 mg) was far more economical. (Duh!)