Modulación inmuno endócrina y metabólica en la infección por Trypanosoma cruzi
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Parasite burden in the heart of infected galectin-3 deficient mice was not significantly different from wild type mice, but in contrast, showed a dramatically reduction in the
Chagas’ disease caused by the intracellular protozoan Trypanosoma cruzi is the most important parasitic infection in Latin America affecting several million persons. None-
We evaluated the satDNA and kDNA OligoC-TesTs in a multi-cohort study with 187 chronic Chagas patients and 88 healthy endemic controls recruited in Argentina, Chile and Spain and
cruzi infection and ET-1 cooperatively activated the Ca 2+ / calcineurin (Cn)/nuclear factor of activated T cells (NFAT) signaling pathway in atrial myocytes, leading to COX-2
This study supports parasite variability as a cause for the diverse cardiac outcomes observed in Chagas disease, and suggests that endothelial cells could