MicroRNA-3148 Modulates Allelic Expression of Toll-Like Receptor 7 Variant Associated with Systemic Lupus Erythematosus

SAA1 remains stable in patients with a favorable outcome, while plasma protein concentrations in the unfavorable outcome group increased at 72 h after the injury (Figure 2A)..

A common concern with self‐reported data, like SAH, is the existence of omitted individual characteristics that 

Toll-like receptor-4 (TLR4) signaling augments chemokine- induced neutrophil migration by modulating cell surface expression of chemokine receptors. Overexpression of G

We evaluated TLR2-mediated IFN-β production in peritoneal macrophages and the Raw264.7 cell line, observing that TLR2 ligands induce IFN-β although at lower levels than TLR4

Treatment of AngII-infused mice with anti-TLR4 antibody improved these structural parameters (Figure 2A-D); the treatment also improved the reduced number of smooth

The roles of the TLR4 signaling pathway in the processes underlying inflammatory vascular diseases including atherosclero- sis [14], diabetes [26–28] or pre-eclampsia [29,30] have

AAV9-CnAβ1 decreases the expression of genes associated with heart failure and fibrosis in the remote myocardium post MI – AAV9-CnAβ1 administration at a dose of 3.5e10 VP,

Neurons, astrocytes, microglial or endothelial cells are susceptible to α-Syn aggregates (i.e., by phagocytosis, endocytosis, Toll-like receptor (TLR) stimulation, etc.), which