Basic fibroblast growth factor reduces functional and structural damage in chronic kidney disease

Although hyperglycemia, oxidative stress, and RAAS activation are the driving forces for renal damage associated with diabetes, numerous pieces of evidence point out the key role

5.- DNA damage assays on HL-60 treated cells with different lyophilised beverages and biological compounds showed that CCC, CFCC, FRU and CRB induced DNA fragmentation

In addition, a signif- icant improvement of tubulointerstitial damage was indicated by reduced gene expression of kidney injury molecule-1 28 in diabetic gapoE compared with apoE

Kopple, “Association of dietary phosphorus intake and phosphorus to protein ratio with mortality in hemodialysis patients,” Clinical Journal of the American Society of Nephrology,

Effects of basic fibroblast growth factor on early revascularization and epithelial regeneration in rabbit tracheal orthotopic transplantation. Comparative anatomy, physíology,

Canagliflozin and Renal Outcomes in Type 2 Diabetes and Nephropathy (CREDENCE), Dapagliflozin on Renal Outcomes and Cardiovascular Mortality in Patients with Chronic Kidney

In CCN2(IV)-injected mice, renal MCP- 1 and RANTES gene expression was significantly increased in response

To test the efficacy of telomerase gene therapy in our mouse model of pulmonary fibrosis induced by DNA damage to the lungs (ie.,low-dose bleomycin) and the presence of short