Transmisión intradomiciliaria de Trypanosoma Cruzi por Rhodnius Ecuadoriensis

These results correlated with our previous kinetics of arginase I expression in the hearts of both C57BL/6 and BALB/c mice (19) and indicated that during in vivo infection, arginase

Parasite burden in the heart of infected galectin-3 deficient mice was not significantly different from wild type mice, but in contrast, showed a dramatically reduction in the

Chagas’ disease caused by the intracellular protozoan Trypanosoma cruzi is the most important parasitic infection in Latin America affecting several million persons. None-

We evaluated the satDNA and kDNA OligoC-TesTs in a multi-cohort study with 187 chronic Chagas patients and 88 healthy endemic controls recruited in Argentina, Chile and Spain and

This study supports parasite variability as a cause for the diverse cardiac outcomes observed in Chagas disease, and suggests that endothelial cells could

cruzi as the antigen fraction, in the diagno- sis of Chagas disease, through the analysis of 258 human sera from the suburban area of the city of Santiago de Quer´etaro (Mexico), by

Previous studies on metabolic alterations caused by T. cruzi infection were based only on host enzymatic expression in vitro [24]. Thus our work greatly expands those results

Detection of Trypanosoma cruzi DNA in blood by PCR is associated with Chagas cardiomyopathy and disease severity.. Eur J Heart