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ACCIONES URBANISTICAS 1 Suelo de expansión urbana

In document SUAN OFICINA DE PLANEACION 2001 (página 35-47)

2. COMPONENTE URBANO

2.3 ACCIONES URBANISTICAS 1 Suelo de expansión urbana

 

Having  demonstrated  that  NHNS  contain  p75  positive  cells  and  putative  ENSC,  the   next   stage   in   the   development   of   a   stem   cell   transplantation   technique   was   to   show   that   these   cells   could   be   transplanted   into   aganglionic   distal   hindgut   and   that   any   new   ENS   neurons   found   in   the   transplant   were   generated   within   the   neurosphere,   rather   than   being   pre-­‐existing   neurons   incorporated   into   the   neurosphere  initially.  

 

For  transplantation,  specimens  of  distal  hindgut  were  harvested  from  e11.5  CD-­‐1   mouse   embryos   as   shown   in   Figure   2.7   and   cultured   on   a   semi-­‐permeable   membrane   (Millicell,   Millipore   UK   Ltd,   Watford,   UK).     This   was   done   by   placing   3ml  of  warmed  tissue  culture  medium  in  a  50mm  tissue  culture  dish  and  placing   the  culture  plate  insert  within  it.    The  hindgut  was  dissected  free  in  sterile  PBS  in   the   same   way   and   the   caecum   was   removed.     The   hindgut   was   then   removed   using   a   sterile   100µm   pipette   and   placed   on   the   culture   plate   insert.     A   40µl   aliquot   of   tissue   culture   medium   (containing   DMEM-­‐high,   see   Appendix   1)   was   then  placed  on  the  hindgut  sample.    Tissue  culture  medium  was  changed  every  3   days  and  the  aliquot  of  medium  on  top  of  the  hindgut  sample  was  also  changed   by  carefully  removing  it  with  a  sterile  40µl  pipette  and  replacing  the  medium.    

Single   human   neurospheres   that   had   been   cultured   for   a   minimum   of   28   days   were   apposed   to   the   proximal   end   of   the   isolated   hindgut   using   a   100µl   sterile   pipette,  as  shown  in  Figure  2.7.    As  controls,  gut  explants  were  cultured  without   neurospheres  and  explants  cultured  with  the  caecum  still  attached,  as  described   in   Figure   2.7A.     Specimens   were   fixed   and   processed   for   immunohistochemistry   after  8  days  in  culture.  

 

Tissue  culture  specimens  were  fixed  by  removing  the  tissue  culture  medium  and   gently   washing   three   times   in   PBS.     All   specimens   were   fixed   in   4%   (w/v)   paraformaldehyde   in   PBS   for   1   h   at   room   temperature   and   then   rinsed   3   times   with   PBS.     They   were   then   immersed   in   20%   (w/v)   sucrose   for   1   h   prior   to   embedding   in   7.5%   (w/v)   gelatine   in   15%   (w/v)   sucrose.     Embedded   specimens   were  frozen  at  -­‐80oC  in  isopentane  and  7μm  serial  sections  cut  using  a  cryostat.    

Immunohistochemistry   was   performed   for   PGP9.5   and   S100   as   described   previously.     To   identify   human   cells   in   the   embryonic   mouse   gut,   mouse   anti-­‐ human  ribonucleoprotein  antibody  (HRNP,  Chemicon)  1:50  was  used  with  Texas   Red-­‐conjugated   goat   anti-­‐mouse   IgG   1:200   (Abcam   Ltd,   Cambridge,   UK).     To   assess   whether   cells   were   generated   within   the   neurosphere,   human   neurospheres  were  incubated  with  10  mM  BrdU  for  18  h  before  transplantation.     Incorporated  BrdU  was  detected  by  a  2  h  incubation  with  mouse  anti-­‐BrdU  (Dako)   diluted   1:25   followed   by   rinsing   in   PBS   and   a   further   2   h   incubation   with   Texas   Red-­‐conjugated   goat   anti-­‐mouse   IgG   (Abcam)   diluted   1:200.     Dual   labelling   was   performed  as  before.    

 

2.5.2  Results  

 

2.5.2.1    Growth  of  NHNS  

 

derived   neurospheres,   there   was   no   statistically   significant   difference   in   size   between  human  and  mouse  neurospheres  when  the  data  for  all  time  points  was   aggregated   (two-­‐tailed   t=test,   p=0.065)   or   when   individual   time   points   were   considered  separately  (Kruskal-­‐Wallis  1-­‐way  ANOVA  with  Dunns  post  test,  p>0.05   for  all  time  points.  

 

Table  2.3:    Yield  and  size  of  EMNS  and  NHNS      

      EMNS   NHNS   Day   Neurosphere   number   Neurosphere   volume     (µm3  x103)   Neurosphere   number   Neurosphere  volume   (µm3  x103)   0   0   0   0   0   7   20   72 +/- 19 18   39 +/- 9 14   53   702 +/- 146 21   199 +/- 74 21   66   671 +/- 80 48   536 +/- 120 28   70   1190 +/- 364 52   783 +/- 100        

Figure 2.8 Growth of human neurospheres

A

B

C

D

E

Figure 2.8 Growth of NHNS

The growth of human neurospheres. Phase contrast micrographs of: (A) Single cell suspension of human cells at D0. (B) Neurospheres at D7 of culture. (C) Neurosphere at D14 of culture. Note neurite outgrowth from the neurosphere (arrowhead). (D) Neurospheres at D21 of culture. Although neuro- sphere size had increased, there also many smaller cell clusters and individual cells present. (E) Neuro- spheres at D28 of culture. Neurosphere size has increased to steady state and there are fewer indi- vidual cells surviving within the culture. (F) Scatter plot (median and range) showing size of NHNS neuro- spheres in comparison with EMNS at the same time- point. Human neurospheres tended to be smaller than their embryonic mouse counterparts, but this

2.5.2.2    Cell  marker  expression  of  single  cell  suspension  and  NHNS  

 

Immunofluorescence   of   the   single   cell   suspension   derived   from   digesting   the   circular  and  longitudinal  muscle  layers  is  shown  in  Figure  2.9.    This  demonstrates   that   within   the   digested   sample   there   are   cell   positive   for   human   nuclei,   p75,   PGP9.5   and   S100.     The   majority   of   the   cells   however   are   positive   for   SMA,   as   would   be   expected.     There   were   few   cells   positive   for   p75   and   relatively   more   positive  for  PGP9.5  and  S100B.    This  raises  the  possibility  that  there  may  be  p75   positive  ENSC  present  within  the  sample.  

 

After   30   days   in   culture,   Immunohistochemistry   confirmed   the   presence   of   the   neural   crest   cell   marker   p75   (Figure   2.10A)   as   well   as   glial   and   neuronal   cell   markers   within   mature   neurospheres   (Figure   2.10B   and   C).     BrdU   incorporation   demonstrated   proliferating   cells   that   tended   to   be   uniformly   distributed   throughout  the  neurospheres,  in  contrast  to  mouse  neurospheres  (Figure  2.10D).   Dual   labelling   with   PGP9.5   and   BrdU   showed   the   presence   of   cells   positive   for   both  BrdU  and  PGP,  indicating  that  new  neuronal  cells  had  been  generated  from   actively   dividing   precursors   within   the   neurosphere   (Figure   2.10D   inset,   arrowhead).    Immunofluorescent  labelling  for  neurotransmitters  commonly  found   in  the  ENS  showed  the  presence  of  NOS  (Figure  2.10E),  ChAT  (Figure  2.10F),  VIP   (Figure   2.10G),   SP   (Figure   2.10H)   and   CGRP   (Figure   2.10I)   within   the   neurospheres.   No   TH   immunoreactivity   was   seen   and   the   proportion   of   cells   staining  for  NOS  and  ChAT  was  greater  than  that  for  the  other  neurotransmitters.    

There   appeared   to   be   a   positive   reaction   to   c-­‐Kit   in   the   primary   neurospheres   (Figure   2.11A   and   B)   but   a   similar   pattern   of   staining   was   seen   on   the   negative   control  (Figure  2.11C).    c-­‐kit  and  PGP9.5  dual  labelling  of  transplanted  NHNS  (for   transplantation   and   culture   method,   see   section2.5.1.4)   demonstrated   that     defined   c-­‐kit   staining   appeared   to   appear   within   the   neurosphere   at   the   peripheries   (Figure   2.11D).     It   therefore   seems   likely   that   there   are   some   c-­‐Kit   reactive   cells   within   the   neurosphere   but   the   results   presented   here   cannot  

2.5.2.3    Demonstration  of  clonality  of  NHNS  

 

Despite  multiple  attempts  at  demonstrating  clonality  of  NHNS  in  a  similar  way  to   that   described   for   mouse   neurospheres(Almond   et   al.,   2007),   no   neurospheres   formed   under   these   conditions.     However,   when   NHNS   were   cultured   at   clonal   density,   then   new   neurospheres   began   to   form   (Figure   2.12A   and   B).     These   neurospheres  were  very  low  in  number  (typically  1  or  2  per  90mm  Petri  dish)  but   displayed   the   typical   morphology   of   NHNS   and   produced   neurite   outgrowth   (Figure  2.12C  and  D).      

 

2.5.2.4    NHNS  can  be  transplanted  into  embryonic  mouse  distal  hindgut  

In document SUAN OFICINA DE PLANEACION 2001 (página 35-47)

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