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Identificación de proteínas que interaccionan con repetidos CA

6. Resultados y Discusión

6.4. Identificación de proteínas que interaccionan con repetidos CA

Cerebral Edema: intracellular & extracellular types

Intracellular Edema: ↓serum Na (SIADH), dysfxnal Na/K ATPase pump

(global hypoxia)

Extracellular Edema: ↑vessel permeability, meningitis, metastasis Respiratory Acidosis, Hypoxemia: ↑cerebral vessel permeability,

enhance cerebral edema

Papilledema: sign of cerebral edema

Intracranial HTN: papilledema, bradycardia, projectile vomiting, HTN Pseudotumor Cerebri

o ↑ICP w/out evidence of tumor or obstruction o Most common in young obese women o ↓CSF resorption in arachnoid granulations o Headache, blurry vision, diplopia

Cerebral Hernation: complication of intracranial HTN

Subfalcine Herniation: compression of anterior cerebral artery Uncal Herniation

o compression of CN III, Posterior Cerebral Artery, parasympathetic fibers

o eye deviated down & out, mydriasis

Tonsillar Herniation: coning of cerebellar tonsils, cardiorespiratory arrest Hydrocephalus: enlargement of ventricles

CSF: produced by choroid plexus, reabsorbed by arachnoid granulations Communicating Hydrocephalus: ↑production CSF, ↓reabsorption CSF Noncommunicating Hydrocephalus: obstruction CSF outflow into

ventricles

Sylvius Aqueduct Blockage: most common cause of hydrocephalus in

newborns

Hydrocephalus

o Children: ventricles dilate & enlarge head circumference o Adults: no ↑ in head size, dementia, gait disturbance, urinary

incontinence

Hydrocephalus ex vacuo: dilated ventricles 2° to brain atrophy Normal Pressure Hydrocephalus

o dilated ventricles + triad—dementia, urinary incontinence, wide- based gait

o potentially reversible cause of dementia w/ shunting

o Wide-Based Gait/Urinary Incontinence: stretching of sacral motor fibers

o Dementia: stretching of limbic fibers

Developmental Disorders

Neural Tube Defects: failure of fusion of lateral folds of neural plate,

↑AFP

Maternal Folate level must be adequate BEFORE pregnancy Anencephaly: absence of brain, maternal polyhdraminos

54  Spina Bifida Occulta: dimple/tuft of hair overlying L5-S1

Meningocele: cystic mass w/ meninges

Meningomyelocele: cystic mass w/ meninges & spinal cord

Arnold-Chiari: caudal extension medulla/cerebellar vermis through

foramen, hydrocephalus, meningomyelocele, syringomyelia

Dandy-Walker: partial/complete absence of cerebellar vermis, cystic

dilation of 4th ventricle, hydrocephalus

Syringomyelia

o degenerative dz of spinal cord, usually cervical cord o cervical cord enlargement, fluid-filled cavity

o ↓pain/temp sensation in hands, loss intrinsic hand muscles o MRI shows cervical enlargement & cavity

Phakomatosis: neurocutaneous syndromes Neurofibromatosis

o Auto dom, incomplete penetrance

o Both Type 1 & 2—café au lait macules, neurofibromas

NF1

o optic gliomas, Lisch Nodules, axillary/inguinal freckling

o associations: pheochromocytoma, Wilm’s Tumor, CML (juvenile)

NF2: bilateral acoustic neuromas, juvenile cataracts, meningiomas Tuberous Sclerosis

o Auto dom

o Mental retardation, hamartomas in brain, kidneys

o Key Findings: seizures, mental retardation, angiofibromas, ash leaf lesions

o Rhabdomyoma of Heart: highly predictive of tuberous sclerosis

Sturge-Weber Syndrome: vascular malformations of face, ipsilateral

arteriovenous malformation in meninges in some pts

Head Trauma

Coup Injuries: site of impact

Contrecoup Injuries: opposite site of impact

Epidural Hematoma: temporoparietal skull fx, Middle Meningeal A. tear CT Scan: imaging test of choice

Subdural Hematoma

o Venous bleed between dura & arachnoid membranes o Most often caused by trauma, ↑ risk w/ cerebral atrophy o Tear of bridging veins producing venous blood clot o CT Scan: imaging test of choice

CNS Vascular Disorders Global Hypoxic Injury

o Hypotensive episodes, chronic CO poisoning

o Complications: cerebral atrophy, watershed infarcts, stroke

Red Neurons: apoptotic neuron

Hypoglycemia: ≈effect on brain as global hypoxia Strokes: ↑ incidence w/ age

Atherosclerotic Stroke

o Most common overall stroke, ischemic type

o Pale infarction extending to periphery of cerebral cortex

o Most occur in MCA distribution

o Infarction w/ liquefactive (NOT coagulative) necrosis

TIA: transient neurologic deficit lasting <24hr, microembolization of

plaque material

Amaurosis Fugax: temporary loss of vision, embolic material trapped at

bifurcation of retinal vessels

MCA Stroke: contralateral paresis/sensory loss in face/upper extremity,

head/eyes deviate to side of lesion

ACA Stroke: contralateral paresis/sensory loss in lower extremity

Embolic Stroke

o Ischemic type of stroke dt embolism

o Hemorrhagic infarction extending to periphery of cerebral cortex

Intracerebral Hemorrhage

o complication of HTN, rupture of aneurysm

o basal ganglia most common location

Rx HTN ↓ the incidence of stroke by more than 40%

Subarachnoid Hemorrhage

o Rupture of congenital Berry Aneurysm

o Severe occipital headache, described as “worst headache ever”

Berry Aneurysms: jxn of communicating branch w/ main cerebral

artery

Lacunar Strokes

o microinfarction <1cm

o hyaline arteriolosclerosis dt HTN/diabetes

55 CNS Infections

CNS Infection: most dt sepsis Meningitis

o inflammation of pia mater

o ↑CSF protein—bacterial, fungal & some viruses o ↓CSF glucose—bacterial, fungal

Bacterial Meningitis: majority of organisms originate in nasopharynx Viral Meningitis: most often transmitted by fecal-oral route

Encephalitis: inflammation of brain, headache, drowsiness, coma Cerebral Abscess: hematogenous, contiguous spread

Demyelination Disorders

Demyelination: destruction normal myelin/oligodendrocyte, abnormal

myelin

Multiple Sclerosis

o Most common demyelinating disease

o CD4 T cells react against self antigens in myeline sheath, cytokines activate macrophages that destroy myelin o Genetic factors & environmental triggers

o Blurry vision dt optic neuritis—MS most common cause

Demyelinating Plaques: white matter looks like gray mater

Sensory Dysfxn: paresthesis, loss pain/temperature/vibratory sensation Upper Motor Neuron Dysfxn: spasticity, ↑deep tendon reflexes, muscle

spasm, Babinski, weakness

Autonomic Dysfxn: urge incontinence, sexual dysfxn, bowel motility

dysfxn

SIN: scanning, speech, intention tremor, nystagmus

Bilateral Internuclear Ophthalmoplegia: pathognomonic for MS,

demyelination of medial longitudinal fasciculus

Lab: ↑CSF lymphs, CSF protein, CSF MBP, normal CSF glucose Oligoclonal Bands in High-Resolution Electrophoresis: sign of

demyelination

Central Pontine Myelinolysis: dt rapid IV correction of hypNa00usually in

an alcoholic

Adrenoleukodystrophy: XR, peroxisomal enzyme deficiency in β-

oxidation of fatty acids

Metachromatic Leukodystrophy: LSD, deficiency arylsulfatase A

Krabbe’s Dz: LSD, deficiency β-galactocerebrosidase w/ ↑ in β-

galactocerebroside in lysosomes

Degenerative Disorders Alzheimers

o Most common overall cause of dementia o Sporadic late onset type—most common type o Prevalence ↑s w/ age

o ↑phosphorylated Aβ  neurotoxic

o ↑ density of NF tangles & senile *neuritic) plaques in brain, occipital lobe usually spared

o Prominent early sign—decline in short-term memory

o Presumptive dx w/ mental status testing, r/o all other causes of dementia

Activated GSK-3β: phosphorylates Aβ

o can be converted into amyloid, deposits in cerebral vessels o metabolic product of APP, coded for on chromosome 21

Secretases: β-secretases followed by γ-secretases cleave APP  Aβ Insulin Degrading Enzyme: involved in clearance of Aβ

Apo Gene E, Allele ε4: sporadic early onset AD Activated GSK-3β: hyperphosphorylates tau protein

Neurofibrillary Tangle: hyperphosphorylated tau protein in neuron PIN1 Enzyme: dephosphorylates hyperphosphorylated tau protein,

deficient in some cases of AD

Senile (Neuritic) Plaques: core of Aβ surrounded by neuronal cell

processes w/ tau protein

Amyloid Angiopathy: risk for cerebral hemorrhage Confirmation of AD: must be made at autopsy Parkinsonism

o Alteration in dopaminergic pathways involved in voluntary muscle movement

Dopamine: principle NT in nigrostriatal tract Idiopathic Parkinsons Dz

o most common cause of Parkinsonism

o depigmentation substantia nigra neurons, ↓dopamine o Clinical: rigidity, resting tremor, bradykinesia

o Expressionless face, blepharospasm, seborrheic dermatitis

56

o Auto dom, trinucleotide repeat disorder

o Atrophy of caudate nucleus, putamen, globus pallidus o Chorea, oculomotor abnormalities

Friedreich’s Ataxia

o Auto rec, trinucleotide repeat disorder, deficiency frataxin o Degeneration Sites: dorsal root ganglia,

posterior/spinocerebellar/corticospinal tracts

o Hypertrophic cardiomyopathy, type I diabetes mellitus

Amyotrophic Lateral Sclerosis

o Degeneration of Lower & Upper Motor Neuron o Atrophy of intrinsic muscles of hand—1st

LMN sign o No sensory Δs, bowel & bladder fxn intact

Werdnig-Hoffmann Dz: Lower Motor Neuron dz in children Toxic & Metabolic Disorders

Wilson’s Dz: cystic degeneration of basal ganglia

Lenticular Nucleus—putamen & globus pallidus in the basal ganglia Acute Intermittent Porphyria

o Urine colorless when 1st

voided, exposure ot light produces color o Deficiency uroporphyringoen synthase, “bellyful of scars”,

peripheral neuropathy, dementia

o Rx: carbohydrate loading inhibits ALA synthase

Vitamin B12 Deficiency: subacute combined degeneration, dementia Wernicke-Korsakoff Syndrome

o hemorrhage in mammillary bodies

o confusion, ataxia, nystagmus, ophthalmoplegia

Alcoholics receiving IV infusion w/ glucose: supplement IV w/ thiamine

to prevent acute Wernicke’s Encephalopathy

CNS Tumors

Glioblastoma Multiforme: most common 1° CNS tumor in adults Childhood Tumors: cystic astrocytoma & medulloblastoma—both in

cerebellum

Clinical: headache, seizures, intracranial HTN Astrocytoma: most common neuroglial tumor

Glioblastoma Multiforme: grade IV astrocytoma, often crosses corpus

callosum, hemorrhagic/cystic

Meningioma

o Most common benign brain tumor in adults

o Female predominance o Psammona bodies

Ependymoma: 4th ventricle in kids & cauda equina in adults

Medulloblastoma: small cell tumor in cerebellum Oligodendroglioma: frontal lobe calcifications in an adult 1° CNS Lymphoma: occurs in AIDS, EBV-mediated cancer Metastasis: most common brain malignancy

Peripheral Nervous System Disorders

Sensory Δs: demyelination—parethesias, “glove & stocking” distribution Motor Δs: axon degeneration—muscle fasciculations, atrophy

Charcot-Marie-Tooth Dz

o Most common hereditary neuropathy

o Lower legs have “inverted bottle” appearance

Guillain-Barre Syndrome

o Most common acute peripheral neuropathy

o Preceding Infections: M. pneumonia, C. jejuni, viruses o Causes ascending paralysis

o Rx: IV immunoglobulin or plasma exchange

Diabetes Mellitus: most common cause of peripheral neuropathy Idiopathic Bell’s Palsy

o Facial muscle paralysis dt inflammation of CN VII o HSV most common association

Upper Motor Neuron Bell’s Palsy: contralateral weakness lower face,

sparing of upper face

Drugs: vincristine, hydralazine, phenytoin

Vitamin Deficiencies: thiamine, pyridoxine, vitamin B12 Schwannoma: benign Schwann cell tumor