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 periodically monitor for gastric cancer ---

Vit B12 deficiency is common after total or partial

gastrectomy or chronic gastritis

o 2/2 loss of intrinsic factor

Vit B12 is a cofactor in purines & DNA synthesis

ineffective erythropoiesis  megaloblastic anemia

total RBC count & reticulocyte count are low  shiny tongue (glossitis) & pale palmar creases ---

ineffective erythropoiesis is a hallmark of both

Vit B12 & folate deficiency

 delayed nuclear maturation   # of megaloblasts  ineffective erythropoiesis due to  transition to mature RBC &  death of immature precursors  total RBC count & reticulocyte count are low ---

antiviral Rx for influenza: oseltamivir o must be started within 48 hrs for efficacy  confirm Dx: nasal swab for influenza antigens ---  fever, leukocytosis, LUQ pain: splenic abscessleft pleuritic chest pain, left pleural effusion,

splenomegaly

 MCC: Staph, Strep, Salmonella

MC risk factors: infective endocarditis with

hematogenous spread

Rx: ABX + splenectomy

 percutaneous drainage for poor surgical candidate  DDx: malaria, Hodgkin’s, infectious mono, TB ---  anorexia, constipation,  thirst, easy fatigue,

hypercalcemia, smoking Hx: SCC of lung o sCa++mous cell carcinoma

PTHrP  Ca++ resorption from bone &  renal Ca+

+ resorption from distal tubule  CXR: hilar mass

 DDx: small cell carcinoma (a/w ACTH or SIADH), adenocarcinoma (a/w hypertrophic osteoarthropathy) ---  OTC cold meds containing acetaminophen &

phenylephrine can provoke warfarin-associated IC hemorrhage

 acetaminophen potentiates warfarin effects  phenylephrine elevates BP

Rx: prothrombin-complex concentrate (rapid & short-term warfarin reversal) & IV Vit K

(promotes clotting factor synthesis; 12 – 24 hr)  alt Rx: FFP if PCC not available; FFP takes longer

to prepare/administer & more volume infusion ---

VIPoma

feature

s watery diarrhea, weight loss, lethargy,facial flushing, N/V, muscle weakness

labs hypokalemia ( intestinal K+ secretion),

acholrhydria (  gastric acid secretion),

hypercalcemia ( bone resorption), hyperglycemia ( glycogenolysis), secretory diarrhea:  Na+,  osmolal gap

Dx watery diarrhea, VIP > 75 pg/mL

abdominal CT (usually pancreatic tail)

Rx IV fluids, octreotide to decrease diarrhea

 VIP binds intestinal epithelial cells to increase fluid & electrolyte secretion into intestinal lumen  maybe a/w MEN Type I

---  steatorrhea, hepatomegaly, PUD: systemic

mastocystosis

o pruritus, facial flushing, urticaria

---  striatal neuro-degeneration: Huntington’sspongiform encephalopathy: CJD

loss of nigrostriatal DA neurons: Parkinson’sselective loss of cholinergic neurons: Alzheimer’sneurodegeneration of frontal/temporal lobes: Pick’s ---

rationalization: excusing an unacceptable

behavior/emotion in a false, but logically rational way to avoid the true reason for a behavior, to prevent anxiety & protect self-esteem

 can result in a delay in care or difficult Rx course ---

repression: blocking upsetting ideas/impulses

from entering consciousness; blocking inner states

denial: blocking external sensory data

---  acute dyspnea, pleuritic chest pain, tachycardia,

follwed by hypotension, syncope: acute massive PE accompanied by RV dilation & hypokinesis 2/2

occlusion of the pulmonary artery, which  RA, RV, & pulmonary artery pressure

 can cause RBBB & right axis deviation  septal deviation toward LV results in  LV preload

&  cardiac output

PCWP & SVR are not affected

---

hypovolemic shock:  RA, RV, pulmonary artery,

& PCWP;  SVR to maintain organ perfusion

cardiogenic shock:  PCWP & SVR,  C.O.

o  cardiac index,  RA pressure

septic shock: peripheral vasodilation &  SVR,

 RA, pulmonary artery, & PCWP,  C.O. o  mixed venous O2 saturation

---

acute mitral regurgitation as a complication of

o flash pulmonary edema & crackles

---  MCC of 20 HTN in children, to-&-fro bruit @ CVA:

fibromuscular dysplasia

 angiogram: “sting of beads”

---

viral meningitis: self-limited inflammation of

leptomeninges cause by viral infection  MCC: echovirus or coxsackie virusMC in infants; incidence decreases with age  prodrome constitutional & URI symptoms; next

36 – 48 hrs  high fever, headache, irritability, &

nuchal rigidity

 WBC (lymphocyte predominance), normal

glucose, mildly elevated protein

 Rx: supportive; resolves in 7 – 10 days

 DDx: bacterial (neutrophils,  protein,  glucose), TB meningitis (lymphocytes,  protein,  glucose) ---  hemolytic anemia, cytopenias, hypercoagulability:

PND

 Dx: RBC CD55 & CD59 testing

---  oral corticosteroids: Rx acute asthma exacerbation ---

Management of GERD

 no cancer risk factors or alarm symptoms  PPI o if refractory, try another PPI or  to 2x daily  male, age > 50, symptoms for > 5 yrs, or cancer risk

factors or alarm symptoms  upper endoscopy o if no evidence of esophagitis  manometry

GI alarm symptoms

hematemesis, persistent vomiting, anemia, melena, weight loss, dysphagia/odynophagia ---

intrauterine fetal demise: in utero fetal death

after 20 wks gestation, before labor onset o Dx: USS

next: coagulation profile to detect incipient DIC o retention of dead fetus can cause chronic

consumptive coagulopathy due to gradual release of thromboplastin from the placenta

low/normal fibrinogen may indicate early signs of

consumptive coagulopathy if a/w  PLT count,  PT, PTT, or fibrin split products

o fibrinogen is usually higher in pregos

 if any coagulation derangements  prompt delivery  if normal coagulation parameters…

o watchful expectancy or labor induction o expectant management complications:

chorioamnionitis & DIC

---

Congenital hypothyroidism

etiology thyroid dysgenesis (MCC) features most are asymptomatic,

jaundice, lethargy, hoarse cry, dry skin,

poor feeding, constipation, large tongue Dx  TSH,  free T4, via newborn screening Rx levothyroxine

---

maternal Graves’: transplacental passage of

TSH receptor antibodies  congenital Grave’s: poor feeding, jitteriness, tachycardia, weight loss ---

synchronized cardioversion: Rx a-fib, a-flutter,

stable monomorphic v-tach

temporary transvenous pacemaker: sick sinus

syndrome, 2nd or 3rd degree heart block

---

duodenal hematoma: MCC direct blunt abdominal

trauma; MC in children  Dx: CT with oral contrast

 #1 Rx: NG suction & parenteral nutrition; spontaneous resolution in 1 – 2 wks

 #2 Rx: surgical removal if conservative Rx fails ---

Vaccines for pregos

all pregos Tdap & inactivated influenza

special

circumstances Hep A & B (high risk)Pneumococcus (2nd & 3rd trimester if high risk),

H. influenza (if asplenic), Meningococcus (if high risk), Anti-D Ig (if Rh-negative mother) not

recommended

HPV, MMR, Varicella, smallpox, intranasal influenza

**avoid conception for 4 wks

unvaccinated pregos with confirmed rubella

exposure are offered termination of pregnancy

or Rx prego with IV Ig

 serology not needed if documentation of vaccine ---

Evaluation of nipple discharge

 unilateral  likely malignant

Dx: mammogram +/- USS, surgical evaluation  bilateral  color of discharge??

o bloody or serous  mammogram o milky, non-bloody…

palpable mass or skin changes??

 yes  mammogram

 no  likely physiologic discharge

physiologic galactorrhea

o painless, B/L milky-brown-gray-green o MCC: hyperprolactinemia

 Dx: pregnancy test, TSH & prolactin o Dx: pituitary MRI if prolactin is elevated ---  frequent exposure to ototoxic agents

(aminoglycosides for P. aeruginosa) for CF Rx can cause sensorineural deafness

---  back pain, constipation, anemia, renal dysfunction,

 ESR: MM

o hypercalcemia causes polyuria,

constipation, confusion, or acute pancreatitis ---  acute onset polyarticular & symmetric arthritis

that resolves within 2 wks: viral arthritis 2/2

Parvovirus B19

o adults in frequent contact with children  morning stiffness < 30 min, no joint swelling

o MCP, PIP, wrist, knees, ankles

transient aplastic anemia

 Dx: anti-B19 IgM antibodies

 Rx: resolves spontaneous in 2 – 3 wks, no Rx  DDx: RA (arthritis > 6 wks), SLE, rheumatic fever ---  progressive dyspnea, tricuspid regurgitation,

peripheral edema: pulmonary HTN due to LV

 mean pulmonary arterial pressure ≥ 25 mmHg @ rest (normal ≤ 20 mmHg)

#1 initial Rx: furosemide & ACE-I

---

Impetigo

Non-bullous Bullous-type

MCC S. aureus or

Group A Strep S. aureus

feature s localized painful, non-itchy pustules & honey-crusted lesions; lymphadenopathy rapidly enlarging

flaccid bullae with

yellow fluid,

“collarette” of scale @ periphery of lesion

Rx topical mupirocin oral ABX (cephalexin,

dicloxacillin, clinda)  predisposing factors: warm, humid climates,

poverty, crowding, pre-existing skin trauma,

atopic dermatitis

 prevention: hand-washing

 complications: post-strep GN, rheumatic fever ---  melena, episodic gnawing, nocturnal abdominal

pain relieved by eating: duodenal ulcer  unopposed acid enters duodenum causing pain  high a/w H. pylori infection

 Rx H. pylori-associated PUD: triple therapy with

PPI + amoxicillin + clarithromycin

 Rx PUD only: PPI or H2 blocker

 DDx: gastric ulcer (pain worse with eating) ---  CHF 2/2 restrictive cardiomyopathy, bibasilar rales,

right-sided pleural effusion: hemochromatosis  LV volume is normal with symmetric thickening  Rx of hemochromatosis (phlebotomy) can

reverse the cardiac dysfunction

 DDx: amyloidosis, sarcoidosis, & scleroderma all cause non-reversible cardiac dysfunction ---

Stages of syphilis

primary painless chancre

Dx: spirochetes on darkfield microscopy Rx: IM benzathine PCN G 1x

PCN-allergic: oral doxycycline x14 days

secondar y

diffuse rash (+ palms/soles), hepatitis,

condyloma lata, lymphadenopathy

Rx: IM PCN G 1x

PCN-allergic: oral doxycycline x 14 days latent asymptomatic

Rx: IM PCN G 3x doses

PCN-allergic: doxycycline x 28 days tertiary Tabes dorsalis, Argyll-Robertson pupil,

dementia, aortic insufficiency, gummas Rx: IV PCN G x14 days

PCN-allergic: ceftriaxone x 14 days pregos intrapartum transmission

Rx: PCN G

PCN-allergic: desensitize to PCN  congenital syphilis: failure to thrive, meningitis,

seizures, congenital defects

---  MCC of complicated influenza pneumonia: S. aureus  MC in hospitalized, nursing homes, IVDA, CF  Gram+ cocci in clusters

 Rx: anti-staphylococcal ABX

---

TNM staging is most important prognostic factor

in breast cancer

---  depressed patients must be closely monitored for

suicidality within the first few weeks of initiating

pharmacotherapy; more likely to act on impulses as motivation & initiative improve before a depressed outlook

---  tachypnea, retractions, grunting, nasal flaring, & cyanosis @ birth: respiratory distress syndrome  MCC are immature lungs & surfactant deficiency  risk factors: prematurity, maternal diabetes,

cesarean without labor

maternal diabetes delays surfactant maturation

due to fetal hyperinsulinemia, which antagonizes cortisol, thus blocks sphingomyelin maturation  CXR: “ground glass” opacities, air bronchograms ---

Cholesterol embolism (atheroembolism)

risk

factors cardiac catheterization or angiography; with comorbid hypercholesterolemia,

DM Type II, HTN feature

s

livedo reticularis, blue toe syndrome,

acute kidney injury, amaurosis fugax,

Hollenhorst plaques, intestinal ischemia

Dx  Cr, eosinophilia, hypocomplementemia,

eosinophiluria

confirm Dx: skin or renal Bx (biconcave, needle-shaped clefts within occluded vessel) ---  patient undergoing coronary angiography are at

risk for contrast-induced nephropathy  U/A: muddy-brown granular & epithelial casts  Rx: benign, resolves in 3 – 5 days

---

pseudocyesis is a form of conversion disorder

 normal endometrial stipe, pregnancy test negative  Rx: psych evaluation

 DDx: missed abortion (intrauterine collapsed gestational sac, +pregnancy test), ectopic (adnexal mass & empty uterus, +pregnancy test) ---

neoadjuvant therapy: given before standard Rx

adjuvant therapy: given in addition to standard Rx

induction therapy: initial dose of treatment to

rapidly kill tumor cells & induce remission

consolidation therapy: given after induction Rx

with multi-drug regimen to further reduce tumor

maintenance therapy: given after consolidation Rx

to kill residual tumor cells; keep patient in remission

salvage therapy: Rx for a disease when standard

Rx fails (radiation for PSA recurrence after radical prostatectomy)

---

cirrhosis increases risk for HCC, but HCC does

not  risk of cirrhosis

Management of cirrhosis

 periodic surveillance of LFTs, INR, albumin, etc  compensated: asymptomatic or vague symptoms

 USS for HCC +/- AFP; every 6 months  decompensated: jaundice, pruritus, upper GI bleed;

assess complications

o varices: start non-selective β-blockers,

repeat EGD annually**

o ascites: dietary Na+ restriction, diuretics,

o hepatic encephalopathy: lactulose, Rx

underlying cause

---  childhood Hx of recurrent renal stones, +family Hx,

+urinary cyanide nitroprusside screening test, & hexagonal crystals on U/A: cystinuria

 defective transport of dibasic amino acids (Lys, Arg)  poor solubility of cysteine leads to renal stones ---  persistent abdo pain or dyspepsia post-op or years

after cholecystectomy: post-cholecystectomy

syndrome

 due to biliary or extra-biliary causes

 elevated ALP, mildly abnormal ALT/AST, & dilated common bile duct on USS

 MCC: common bile duct stones or sphincter dysFx  Dx: endoscopic USS, then ERCP or MRCP ---

Management of Cushing’s

#1 test: ACTH levels & urinary cortisol  low ACTH  adrenal CT

o MCC: exogenous glucocorticoids o exclude adrenal adenoma & hyperplasia  normal/high ACTH  pituitary MRI

o mass > 6 mm  dexamethasone supp test  no mass or < 6 mm  inferior petrosal

sinus sampling (invasive)

 MCC: ACTH-producing pituitary adenoma, ectopic ACTH, or ectopic CRH

ectopic ACTH: rapid onset HTN, hypokalemia,

proximal muscle weakness, metabolic alkalosis, hyperglycemia; less likely to have central obesity, moon facies

---

Prevention of recurrent nephrolithiasis

dietar

y fluids (produce > 2 L urine/day)reduce Na+, reduce protein, reduce oxalate,

normal Ca++ intake, increase citrate

Rx thiazides (passive Ca++ reabsorption),

potassium citrate (urine alkalization),

allopurinol (for uric acid stones)  reduced dietary Ca++ leads to  oxalate

absorption in the gut  excreted into urine & binds urinary Ca++  Ca++ oxalate stones ---

 fever, odynophagia, dysphagia, drooling, stiff neck, muffled voice, inability to extend neck & widened prevertebral space: retropharyngeal abscess  MC in age 6 months – 6 yrs

 prodrome URI  direct spread of infection

 polymicrobial (Group A Strep, S. aureus, anaerobes)  Dx: CT with contrast

 complications: airway compromise, bacteremia, carotid artery rupture, jugular venous thrombosis ---  young adult with cirrhosis, neuropsych symptoms,

& Kayser-Fleischer rings: Wilson’s (aka hepatolenticular degeneration)

 AR inheritance

 resting tremor, muscle rigidity, slurred speech, depression, paranoia, catatonia

 impaired ceruloplasmin secretion, thus decreased secretion of copper in biliary system

Cu++ deposition in liver, basal banglia, cornea  also a/w Fanconi’s, hemolytic anemia, neuropathy  Dx: liver biopsy, low ceruloplasmin, slit lamp,

 urinary Cu++ excretion

#1 Rx: D-penicillamine or trientine

o oral zinc prevents copper absorption

 #2 Rx: liver transplant

---  liver disease, hyperpigmentation, arthropathy,

diabetes, impotence, dilated cardiomyopathy:

hemochromatosis

---  productive cough, hemoptysis, recurrent fevers,

recent travel: pulmonary TB

 chest CT: upper lobe cavitary lesions with surrounding alveolar infiltrates

---  MC benign cutaneous vascular tumor in adults:

cherry angioma (aka senile hemangioma)

 age 3rd – 4th decade, increases with age

 sharply circumscribed areas of congested capillaries & post-capillary venules in papillary dermis

 benign, no Rx required

---  MC benign vascular tumor in children:

superficial infantile (strawberry) hemangioma

 red, demarcated plaques that blanch  grows rapidly in first 1 – 2 yrs of life, then

spontaneously regress in childhood

Rx: propranolol if at risk for complications ---

spider angiomas are estrogen dependent

 MC in pregos, OCP use, cirrhosis (hyperestrogen)  dilated cutaneous arterioles with a central papule

& radiating blanching capillaries

---  recurrent sinopulmonary & GI bacterial infections

in adults may indicate humoral immunity defect o a/w food allergies & autoimmune disease  Dx: quantitative serum Ig levels

---

NSAIDs can cause SIADH

o NSAIDs potentiate the action of ADH  hypotonic hyponatremia with euvolemia  Dx: r/o hypothyroidism & adrenal insufficiency  Dx: urine & plasma osmolality

 low plasma osmolality with high urine osmolality  serum uric acid is low due to  urinary excretion

& hemodilution

---

mineralocorticoid deficiency: hypotonic

hyponatremia with hypovolemia; elevated K+ ---

nephrotic syndrome & advanced renal failure:

hypotonic hyponatremia with hypervolemia ---   incidence of carpal tunnel syndrome in pregos

2/2 estrogen-mediated depolymerization of ground substance, causing interstitial edema

initial Rx: wrist splinting

 #2 Rx: NSAIDS (risk of miscarriage), local corticosteroid injection, surgical decompression ---

Urinary incontinence

Stress loss of urethral support, intraabdominal pressure exceeds urethral sphincter leaking with cough, sneeze, laugh, lifting Urge detrusor overactivity

sudden, overwhelming, frequent need Overflow impaired detrusor contractility,

constant involuntary dribbling & incomplete emptying

epidural anesthesia: block afferent & efferent

nerves from the bladder; can cause overdistension  post-partum urinary retention: indwelling catheter

for 24 hr to decompress the bladder

---  ankylosing spondylitis & IBD (UC) are a/w

HLA-B27; may occur together

both are +p-ANCA, despite absence of vasculitis ---

infantile colic: crying ≥ 3 hr/day, ≥ 3 days/wk,

for ≥ 3 wks; typically evenings

o resolves spontaneously by age 4 months  Rx: calming techniques, reassurance

---

misoprostol: synthetic prostaglandin, used with

mifepristone to terminate pregnancies ≤ 49 days

---  cyanosis & respiratory distress immediately after

birth, scaphoid-abdomen: congenital

diaphragmatic hernia

 XR: displaced cardiac silhouette, gasless abdomen ---  acute gout Rx: NSAIDs (indomethacin),

colchicine, corticosteroids  prophylactic Rx: allopurinol

---

male pubertal gynecomastia resolves within a few

months – 2 yrs without intervention; reassurance ---

antiphospholipid antibody syndrome promotes

arterial/venous thromboses & recurrent spontaneous abortions

o thrombocytopenia & prolonged PTT  prophylactic Rx: LMWH & low-dose ASA ---  leukopenia & thrombocytopenia in SLE is due to

peripheral immune-mediated destruction

o pancytopenia is common with SLE

---

alcoholic hepatitis: AST:ALT > 2; < 300 IU/L

o also  GGT & ferritin (acute phase reactant) ---

dermatomyositis is an idiopathic inflammatory

myopathy with immune-mediated muscle injury that can be due to a paraneoplastic syndrome from malignancy (lung cancer)

 Dx: muscle biopsy

---

Achilles tendon reflex can be decreased or

absent with age

 lower extremity weakness,  rectal tone, bowel/bladder incontinence, & brisk LE DTRs: possible spinal cord compression

---

ovulatory phase: cervical mucus is profuse,

clear & thin, stretches 6 cm, exhibits “ferning”, & pH > 6.5 (more basic than usual)

o ideal for spermatozoa entering uterus

 pre- & post-ovulatory: scant, opaque, & thick mucus ---  Hx of pelvic surgery or irradiation can develop a

urinary fistula

 Dx: intravenous pyelography

---  young male with lower abdominal pain, tenesmus,

bloody diarrhea with acutely worsening fever, leukocytosis, hypotension, & tachycardia:

ulcerative colitis complicated by toxic megacolon

 may be the first presentation of UC

Dx: abdominal XR with colonic distension > 6 cm  + 3 signs…

o fever > 380C, HR > 120, leukocytosis, anemia  Rx: bowel rest, NG tube, steroids (if UC), ABX

o sigmoidoscopy is used to confirm UC, but risk of perforation in toxic megacolon  severe cases: subtotal colectomy & end-ileostomy ---

sulfasalazine: Rx Crohn’s, UC, RA

---  fever, malaise, productive cough, eosinophilia,

hemoptysis: allergic bronchopulmonary

aspergillosis (ABPA)

 hypersensitivity reaction to Aspergillus  MC a/w asthma & CF

---

academia alone does not lead to CNS depression,

but the underlying cause results in lethargy & altered mental status

 CO2 retention due to underlying COPD can lead to CO2 narcosis (PaCO2 > 60 mmHg)

---  DIC depletes clotting factors & 20 fibrinolysis  thrombocytopenia, prolonged PT & PTT,

decreased fibrinogen, schistocytes

---  predominant unconjugated hyperbilirubinemia

o  bilirubin production (hemolysis) o  bilirubin uptake (portosystemic shunt) o abnormal conjugation (Gilbert’s)  predominant conjugated hyperbilirubinemia

o hepatocellular injury

o  bilirubin excretion in bile canaliculi (Dubin-Johnson)

o intrahepatic cholestasis (10 biliary cirrhosis) o extrahepatic cholestasis (biliary obstruction)  normal transaminases & ALP suggest inherited

bilirubin metabolism disorders

 elevated transaminases & normal ALP suggest intrinsic liver disease (viral, hemochromatosis)  elevated ALP suggest intrahepatic cholestasis or

biliary obstruction

---  painless jaundice, fatigue, weight loss, pruritus,

acholic stools with dark urine,  ALP,  GGT:

malignant biliary obstruction

 MCC: adenocarcinoma, cholangiocarcinoma  Dx: USS or CT; ERCP if non-diagnostic

 DDx: acute choledocholithiasis (acute RUQ pain), chronic autoimmune hepatitis, chronic pancreatitis ---  elderly patient with hip fracture should undergo

definitive surgical correction

surgery can be delayed up to 72 hrs to evaluate surgical risk & address unstable comorbidities ---

Ovarian & adnexal torsion

risk factors ovarian mass ≥ 5 cm, reproductive age, infertility Rx with ovulation induction features sudden onset unilateral pelvic pain,

vaginal bleeding is uncommon

Dx β-hCG to exclude ectopic pregnancy,

USS with color Doppler

Rx laparoscopy with detorsion  rotation of ovary around infundibulopelvic

(suspensory ligament) & utero-ovarian ligaments

adnexal torsion: includes fallopian tube

MC right-side: longer ligament

 DDx: appendicitis, ruptured ovarian cyst, endometriosis, tubo-ovarian abscess

---  gradual onset fever, chills, lower abdominal pain,

vaginal discharge: tubo-ovarian abscess

 risk factors: multiple partners, young age, Hx of PID ---

ovarian hyperstimulation syndrome: ovarian

enlargement from multiple cysts with fluid shifts out of the intravascular space

o results in ascites & hypovolemia

complication of gonadotrophin Rx for infertility ---

major depressive disorder (MDD): persistent &

pervasive sadness, self-critical ruminations, & suicidality a/w feeling worthlessness & hopeless ≥ 2 wks

can be diagnosed anytime during acute grief; bereavement period is not an exception  Rx: SSRI & psychotherapy

---

electroconvulsive Rx: refractory to antidepressants

o #1 Rx if not eating/drinking; acutely suicidal, catatonic, or psychotic

---

first-time DVT with a reversible inciting incident

(surgery)

Rx: unfractionated or LMWH within 48 – 72 hrs if hemodynamically stable

o prevents extension of the clot & future clots rather than lysis of the present clot

o streptokinase & tPA: only Rx STEMI ---  low bone mass with normal mineralization:

osteoporosis

 normal serum Ca++, phosphorus, PTH, & ALP

---  defective mineralization of organic bone matrix:

osteomalacia 2/2 Vit D deficiency

 defective mineralization of growth plates in kids:

rickets --- Rickets risk factors  skin pigmentation, exclusive breastfeeding, inadequate sun exposure, maternal Vit D deficiency feature

s craniotabes (“ping-pong ball” skull),delayed fontanelle closure, frontal bossing, costochondral hypertrophy (rachitic rosary)

genu varum

XR metaphyseal cupping & fraying, epiphyseal widening, osteopenia labs  Ca++ & phosphorus,  ALP & PTH Rx Vit D fortified baby food or formula; or

supplementation

---

Vit D deficiency causes marked hypophosphatemia,

 ALP, normal Ca++, secondary hyperPTH  XR:  bone density with thin cortex, “codfish”

vertebral bodies, & pseudofractures (Looser zones) ---  large anterior fontanelle, lethargy, poor feeding,

macroglossia: congenital hypothyroidism  frontal bossing, anterior shin bowing, saddle-nose,

notched/gap teeth: congenital syphilis

 “bucket-handle” fx = classic metaphyseal lesion:

child abuse

---  large tender, soft-tissue mass with localized pain  MC primary malignant bone tumor in children,

MC @ metaphysis of long bones: osteosarcoma