Lytalase is a prostaglandin PGF-2a and, in my opinion, really cool stuff. Where as insulin was the most non-specific anabolic, PGF-2 was the most site-specific anabolic hormone-like chemical Frank used for chemical muscle enhancement. PGF-2 was best utilized site-specific, meaning that it was most effective when injected directly into a muscle for localized growth. (Gee, you think so?)
In short it was perfect for bringing up lagging body parts and contest prep symmetry adjustments. There is a black market preparation that is a form of PGF-2a that is more powerful and in an oil suspension. This allows it to remain at the administration site for a prolonged period exerting its anabolic effects.
There was another reason prostaglandins had to be utilized site-specific. PGF-2 had a brief active-life that reduced effective response periods for total body stimulation... though some did migrate to the vascular system and trigger anabolism through out the body. And this was where problems arose.
Every muscle in the body responds to PCF-2, including smooth muscle tissue such as intestines, and stomach. An injection too near these areas would have resulted in serious cramping. As you know, cramping is a form of contraction. This means that in some cases concerns of "projectile waste" was possible.
Reports showed that in some individuals, flu like symptoms occurred during prostaglandin use. Women mostly were wise enough to avoid PGF-2 use. (Though a few had utilized the drug effectively)
The localized growth induced by site injection PGF-2 triggered highly effective protein synthesis. The few beast who had utilized PGF-2 properly, in respect to the intent of muscular augmentation, added 1 -2 " to their arms in a 3 week period. Some Beasts had to rotated injection sites on a schedule dictated by training for multiple body-part stimulation.
Some did so simply because PGF-2 injections left the target muscle quite sore for a couple of days and the pain hindered intense training. This was not sore like a testosterone suspension injection site. It was far more painful. Kind of like the pain resulting from 15-20 sets of negative-only straight bar curls.
For this reason, Frank did not inject PGF-2 into a target muscle until after it was trained that day. This required Frank to either train first thing in the morning, or to localize sites trained the prior day until after the new day's work-out was complete. Of course multi-sites spread out over multiple days resulted in less growth per part, but an athlete needs to be able to train. Did I mention the soreness some experience?
Another interesting effect of PGF-2 was fat loss. This was fully discussed in the first "Chemical Muscle Enhancement". But to briefly touch on this phenomenon... PGF- 2 triggered a chain of metabolic Action/ Reaction Factors that is in part dependant upon fatty acids. Fatty acids were rapidly released from adipose tissue (fat cells) to fuel these actions. COOL!
"There will always be individuals who claim something is not true due to what someone else has said. Those who say PGF-2 does not induce dramatic effects simply have not used it or failed to procure the real deal."
PGF-2 protocols dramatically increased androgen receptor-site count and site sensitivity. This effect was so dramatic in some beasts that even the elevation in endogenous testosterone production created from HCG use caused muscular pumps so intense that benching an empty bar was enough to bring tears to (some) eyes. These individuals would have been obviously unwise to have utilized AAS with PGF-2. However, "most" advanced athletes did not react this intensely.
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Since PGF-2 had this effect upon androgen receptor-sites, protocols utilizing it was perfect for leading into a Max Androgen Phase or other AAS protocol. This was due to multiple reasons.
First, even lower plasma thresholds of AAS brought significantly better results. Second, the synergistic factors realized when the last 7-1 5 days of PGF-2/insulin use over lapped (layered over) the first 7-15 days of a Max Androgen Phase were quite impressive. There were other reasons this was so effective, but I will wait until the end of this chapter for better understanding.
Frank utilized the properties of insulin to act synergistically with PGF-2. Since PGF-2 was notably protein synthesis magic, a great deal of amino acids (from protein) had to be available at the muscle site when anabolism was triggered. Inadequate availability of nutrients would have produced poor results.
To utilize PGF-2 without assuring building materials would have been like telling the wife and her sister a threesome would rock: Message sent and received, but it is highly unlikely anything good would result. Only very bad things, including a huge waste of money on divorce in most cases. Of course, as I have said countless times prior, some have responded positively to almost any stimulus. We are talking muscle chemistry of course.
Supraphysiological levels of insulin facilitated the metabolization, transport, and storage of supraphysiological levels of macronutrients. Obviously, only because massive amounts of protein, carbohydrates, and fats were first ingested and absorbed.
Unfortunately exogenous insulin use alone will trigger anabolism in all tissues including fat stores. However, when insulin was stacked with a specific-tissue anabolic stimulator such as PGF-2, amazing muscular growth occurred. And in this case, fat stores were reduced in proportion to total calorie needs (metabolic) and ingestion.
Frank always ingested at least 10 grams of carbohydrates or glucose substrates per I.U. of insulin injected. He also avoided over lapping injections of insulin since, due to active-life, it would have induced an up-ramping effect of insulin plasma levels. (Bad idea!) He never went to sleep during a insulin high-activity period and always had a meal before sleeping.
Using days listed as examples, Frank's timing sequence usually followed this schedule:
Day#
#1-7: 7am, 2 pm, 9 pm (trained at 1 pm or between 5-9 pm). Slept at 11 pm.
#8-14: 7am, 11:30am, 4pm, 9pm (trained at 1 0 am or between 7-9 pm). Slept at 11 pm.
#15-21: 7am, 10 am, 1pm, 6 pm, 9pm (trained at 5 pm or moved 6pm injection to 5 pm and trained at 8 pm.)
In so many evaluated cases, even 21 days of continuous insulin use was pushing it. Frank never used any insulin protocol for more than 28 days, or repeated use without at least a 28 day insulin-free period.
"Becoming a diabetic is not a good thing or the goal anyone would seek intelligently. That is the equivalent of cutting a finger off to collect insurance. Let me count your fingers, Frank...now the other 9."