• No se han encontrado resultados

Técnica de Procesamiento y Análisis de Datos

 monitor pain for increased frequency/intensity  Rx: observation, reassurance, massage, heat,

stretching, OTC analgesics

 DDx: osteoid osteoma, osteosarcoma, osteomyelitis ---  sclerotic, cortical lesion with a central nidus of

lucency: osteoid osteoma

 pain worse at night, unrelated to activity  benign, bone-forming tumor

Rx: NSAIDS (suggestive of Dx); spontaneous resolution over several years

---  changing the cutoff point of a quantitative

diagnostic test inversely affects sensitivity &

specificity

 lower the cutoff INCREASES sensitivity & decreases specificity (more true positives)  raising the cutoff DECREASES sensitivity &

increases specificity (more true negatives) ---  recurrent sinopulmonary infections, persistent

diarrhea, oral candidiasis, viral infections: SCID  adenosine deaminase (ADA) deficiency

 Dx: absent lymph nodes & tonsils, lymphopenia, absent thymic shadow on CXR, abnormal T, B, & natural killer cell count by flow cytometry ---

Bruton’s (X-linked) agammablobulinemia:

male infant, asymptomatic until age 6 – 9 months; recurrent pyogenic infections (S. pneumo, H.infl) o decreased IgG, IgA, IgM, IgE, &

absent/decreased B cells

o sinusitis, bronchitis, otitis media, Giardia

CVID presents similar to Bruton’s, but CVID has

less severe symptoms & later onset (age 15 – 35 yrs)

o normal circulating B cells

o decreased IgG, IgA, IgM, IgE

---  thrombocytopenia, eczema, recurrent infections:

Wiskott-Aldrich (“WAITER”)

XR, defective WASP gene

 young boy with eczema, thrombocytopenia, recurrent encapsulated spp infections

o Strep pneumo, N. meningitidis, H. influenza  @ birth: petechiae, bleeding from circumcision,

bruises, bloody stools

 PLT production, small PLTs

low IgM, high IgA & IgE

---

Chronic granulomatous disease (CGD): defective

phagocytes due to NADPH oxidase dysfunction  impaired oxidative metabolism within phagocytes;

defective intracellular killing

recurrent catalase-positive infections (S. aureus)

lymphadenitis, skin abscesses

 Gram stain: neutrophils filled with bacteria  Dx: nitro blue tetrazolium test

---

Chediak-Higashi:  chemotaxis, degranulation,

& granulopoiesis

 mild coagulopathy, pancytopenia

partial albinism, peripheral & cranial neuropathy,

hepatosplenomegaly, infections (S. aureus), progressive lymphoproliferative syndrome  Dx: neutropenia, giant lysosomes in neutrophils  Rx: daily TMP-SMX & ascorbic acid

---

Leukocyte adhesion defect (LAD): failure of

innate host defenses due to defective tethering, adhesion, & targeting of myeloid leukocytes to sites of microbial infection

o absence of neutrophils/pus at infection sites  recurrent bacterial infections of skin & mucosal

surfaces, necrotic periodontitis, gingivitis, delayed umbilical cord separation (> 30 days)  early loss of deciduous & permanent teeth  leukocytosis with neutrophil predominance  cultures: S. aureus, G-negative bacilli

---  increased gastric residual volume, vomiting, &

abdominal distension in a preterm neonate:

necrotizing enterocolitis (NEC)

risk factors: prematurity, very low birth weight  gut immaturity & exposure to bacterial from

enteral feeds result in inflammation & damage to bowel wall

XR: pneumatosis intestinalis (intramural air with “train tracks”) & air in portal vein

 leukocytosis, metabolic acidosis

 Rx: decreased rates of NEC in premature infants who are breastfed

 complications: pneumoperitoneum  DDx: duodenal atresia, pyloric stenosis,

Hirschsprung

---

DDx of delayed passage of meconium

(within 48 hrs) level of meconium

obstruction consistency

Hirschsprung rectosigmoid normal

meconium ileus ileum inspissated

meconium ileus results in microcolonDx: contrast enema

---

Hirschsprung disease should be suspected in a

newborn with failure to pass meconium within 48 hr, in the setting of Down syndrome

 poor feeding, abdo distension, absent air in rectum  failure of neural crest cell migration in the

rectosigmoid; positive “squirt sign” on exam  Dx: rectal biopsy show absence of ganglion cells ---

C3 deficiency predisposes to encapsulated bacteria

 C5 – C7 deficiency: recurrent Neisseria infections  C1 esterase deficiency: hereditary angioedema ---  anticholinesterase toxicity due to

organophosphate poisoning

 bradycardia, miosis, salivation  Rx: atropine & pralidoxime

---

hazard ratio: ratio of an event rate occurring in

treatment vs. control group

 ratio < 1 = treatment group has lower event rate  ratio > 1 = treatment group has higher event rate ---

asymptomatic bacteriuria of pregnancy is

treated with ABX to prevent pyelonephritis, preterm birth, low birth weight, perinatal mortality  risk of pyelonephritis is due to  progesterone

causing smooth muscle relaxation, thus ureteral dilation

screen all pregos @ 12 – 16 wks gestation

Rx: amoxicillin, nitrofurantoin, cephalexin are safe  C/I: tetracycline, fluoroquinolones, TMP-SMX

(interferes with folate metabolism in 1st trimester, & risk of kernicterus in 3rd trimester)

---  strongest indicator for future suicide attempt: Hx

of previous attempt(s)

---

β-blocker A/E: worsening of CHF, bradyarrhythmia,

---

loop diuretics can cause hearing loss &/or tinnitus

ototoxicity occur with high doses, renal failure, or

combo with other ototoxic drugs (aminoglycosides) ---

HCTZ/thiazides can cause photosensitivity,

orthostatic hypotension, hypercalcemia

---  HTN & hypokalemia: 10 hyperaldosteronism  prone to diuretic-induced hypokalemia, resulting

in weakness & muscle cramps

 also metabolic alkalosis, mild hypernatremia  no peripheral edema due to spontaneous diuresis

(aldosterone escape)

screening: morning aldosterone:renin ratio o ratio > 20, with aldosterone > 15 ng/dL

suggests primary hyperaldosteronism  confirm Dx: adrenal suppression testing after

oral saline load

positive result requires adrenal CT  MCC: unilateral adenoma or B/L hyperplasia  Dx: CT scan to confirm unilateral mass; equivocal

results require adrenal venous sampling  U/L adenoma Rx: surgery (#1) or aldosterone

antagonist (spironolactone, eplerenone)  B/L hyperplasia Rx: aldosterone antagonists ---

spironolactone: aldosterone/progesterone/androgen

Rc antagonist

 A/E:  libido, gynecomastia, breast tenderness, menstrual irregularities

eplerenone: selective mineralocorticoid antagonist

---

dehydration is a risk factor for venous thrombosis

due to hemoconcentration

 sudden pleuritic chest pain, cough, dyspnea, hemoptysis: PE

 also tachycardia, tachypnea, & hypoxemia  chest CT: wedge shaped infarction  contrast-enhanced CT: filling defects

 CXR: “Hampton’s hump”, “Westermark” sign  causes transudative & exudative pleural effusion  DDx: TB, PCP, bacterial pneumonia, lung cancer ---

 older patient with rapidly progressive dementia, myoclonus, akinetic mutism, behavior changes:

Creutzfeldt-Jakob disease (CJD)

 triphasic sharp wave complexes on EEG, &/or 14-3-3 CSF proteins

 Dx: brain biopsy show spongiform changes  Rx: supportive; death within one year of onset  DDx: ALS, Lewy body dementia, Alzheimer’s,

NPH, peudodementia

---

scheduled cholecystectomy is indicated for all

symptomatic gallstones (acute pancreatitis), once medically stable due to  risk of recurrent episodes  Rx poor surgical candidate: ursodeoxycholic acid

asymptomatic gallstones should not be treated,

except morbidly obese undergoing gastric bypass or presence of porcelain GB

---

genital HSV eruptions: painful vesicles on an

erythematous base that evolve to shallow, “punched-out” ulcerations/erosions

 Hx of genital HSV: Rx prophylactic acyclovir or valacyclovir @ 36 wks to  risk of outbreak during delivery

neonatal HSV risk factors: maternal infection,

vaginal delivery with active lesions

Rx: c-section for pregos with active genital lesions or prodromal symptoms (burning, pain)

 vaginal delivery only in absence of active lesions ---  burning, localized pain & regional hyperesthesia or

allodynia, in the context of recent cancer therapy:

herpes zoster (shingles)

pain may precede rash by several days

---  acute arthritis of right knee, fatigue, constipation,

polyuria: pseudogout

hyperparathyroidism predisposes to pseudogout

 acute formation of calcium pyrophosphate dehydrate (CPPD) crystals, leading to

chondrocalcinosis (calcified articular cartilage)

 attacks are precipitated by trauma, surgery, illness  Dx: synovial fluid (rhomboid-shaped positively

birefringent crystals)  DDx: gout, septic arthritis

---

hydroxyapatite: osteoarthritis

monosodium urate: gout

calcium oxalate: renal calculi

struvite: renal calculi; UTI of urease spp (Proteus)

---

osteoarthritis: non-inflammatory arthritis

 age > 50, morning stiffness < 30 min

 decreased ROM, stiffness after prolonged rest; anterior hip pain exacerbated by walking  crepitus, no TTP, no warmth of joint

 bony enlargement; Heberden & Bouchard nodes  XR: joint space narrowing, osteophytes,

subchondral sclerosis & subchondral cysts  Rx: weight loss slows OA progression

---  Rx mild/moderate OA pain: acetaminophenRx OA exacerbation: NSAID, intra-articular steroid  Rx OA refractory to NSAID & intra-articular steroid:

colchicine

---  recurrent hemarthrosis & skeletal muscle

hemorrhage after mild trauma: hemophilia A & B  menorrhagia & mucosal bleeding is common in

women with von Willebrand disease (AD) ---  MCC of nephrotic syndrome in children age < 10:

minimal change disease

 periorbital edema in the AM, pretibial pitting, nephrotic range proteinuria, hypoalbuminemia

T-cell mediated injury to podocytes cause

increased permeability to albumin  renal biopsy not required for Dx

Rx: empiric steroids upon suspicion of Dx o 90% have complete remission of proteinuria

renal biopsy indicated in age > 10, or if

unresponsive to steroid Rx; exclude other causes ---  bradycardia, miosis, rhonchi, fasciculations,

salivation, lacrimation, urination, defecation:

organophosphate poisoning

 Rx: removal of clothing & washing of the skin prevents transcutaneous absorption

---  TCA overdose Rx: sodium bicarbonate & EKG ---

bicuspid aortic valve, aortic coarctation, & aortic

root dilation; risk of aortic dissection: Turner’s  ovarian dysgenesis; low estrogen & amenorrhea

results in a high FSH/LH & low inhibin  normal GH levels

---

Social anxiety disorder (social phobia)

anxiety about ≥ 1 social situations, > 6 months  fear of scrutiny, humiliation, embarrassment  marked functional impairment

generalized social anxiety disorder

o Rx: SSRI (paroxetine) & CBT

performance-only social anxiety disorder

o Rx: propranolol 30 - 60 minutes prior & CBT o alt: benzodiazepine (avoid if substance abuse)  Buspiron: Rx GAD only, not social anxiety ---

ischemic cardiac pain can be mistaken for

epigastric pain

 SLE & chronic steroid use are risk factors for accelerated coronary atherosclerosis

exercise stress test without imaging if baseline

EKG is normal

o positive stress test  coronary angiography ---

acute exacerbation of MS Rx: methylprednisone

(high-dose IV corticosteroid)

 long-term steroid therapy provides no benefit & does not prevent future relapses

long-term Rx: beta-interferon can decrease frequency of acute exacerbations & relapses o also glatiramer acetate, IV Ig,

cyclophosphamide, or plasmapheresis ---  MCC of constrictive pericarditis in developing

countries: TB

MCC in the US: viral pericarditis

 pericardial scarring & thickening results in diastolic dysfunction; pericardial calcifications  decreased cardiac output & venous overload

 pericardial knock, pulsus paradoxus, Kussmaul sign, prominent x & y descents

---  CXR: parenchymal nodules (silicosis)

CXR: pleural plaques (asbestosis)

---  hard, irregular, fixed breast mass: malignancyrubbery, firm, freely mobile mass: fibroadenoma  Hx of 1st-degree relative with breast cancer

requires diagnostic (vs. screening) breast imagingpalpable breast mass age < 30: USS

palpable mass age > 30: mammogram, then USS  simple cyst Rx: needle aspiration

 complex cyst/solid mass: image-guided core Bx  suspicious malignancy: core needle biopsy ---  both folate & Vit B12 are involved in conversion

of homocysteine  methionine, thus a deficiency in either results in  homocysteine levels  Vit B12 is involved in conversion of

methylmalonyl-CoA  succinyl-CoA

 Vit B12 deficiency results in methylmalonic acid ---

haptoglobin binds free Hb to form Hb-haptoglobin

complexes removed by the liver   haptoglobin in hemolytic anemias

---

alpha-fetoprotein is elevated in HCC &

testicular germ cell tumors

---  low-grade fever & leukocytosis are common

during the first 24 hr postpartum

 intrapartum & postpartum chills are common

lochia rubra (bloody discharge) is characteristic

postpartum; after 3 – 4 days  lochia serosa (pale)  lochia alba (white/yellow)

foul-smelling lochia suggests endometritis  fever & elevated WBCs beyond 24 – 48 hr requires UTI work-up: U/A, blood cultures ---  blunt deceleration trauma (MVA, fall from > 10 ft),

must rule out blunt aortic injury  high mortality rate

 Dx: CXR  widened mediastinum

 DDx: myocardial contusion (tachycardia) ---  ipsilateral ataxia, nystagmus, intention tremor,

loss of coordination: cerebellar tumor  patient falls/sway TOWARD the lesion side ---  age > 40, personality change, dementia, chorea:

Huntington’s

wide-based gait, steppage gait: Tabes dorsalis  affected arm adducted, affected leg extended; leg

is swung out in a semicircle: hemiparesis/strokewaddling gait: muscular dystrophy

---  risk factor for cervical insufficiency: Hx of

maternal OB trauma, prior GYN surgery (LEEP, cone Bx), multiple gestation, Hx preterm birth, or 2nd trimester abortion

o Dx: transvaginal USS

 risk factors for placental abruption: chronic HTN,

smoking, cocaine use, Hx of maternal trauma, Hx of external cephalic version

 risk factors for uterine rupture: multiparity, Hx

of c-section or myomectomy, adv maternal age, fetal macrosomia

---  abdo pain that refers to one or both shoulders

suggests subdiaphragmatic peritonitis due to irritation of subdiaphragmatic parietal peritoneum  blunt traumatic bladder injuries, bladder dome

is the only region covered by peritoneum, most susceptible to rupture

 MC site of extraperitoneal bladder rupture:

bladder neck

---

unfractionated heparin is preferred over

enoxaparin (LMWH), fondaparinux, & rivaroxaban for severe renal insufficiency  reduced renal clearance  anti-Xa levels,

thus  bleeding risk

monitor unfractionated heparin with aPTTonce therapeutic, initiate warfarin

o warfarin takes 5 – 7 days to be therapeutic; must bridge with heparin

Rivaroxaban has immediate onset of action, does

not require bridging with heparin; but no antidote ---  upper GI bleeding, depressed consciousness level,

& ongoing hematemesis due to esophageal variceal hemorrhage should first be intubated  Rx: endoscopic band ligation or sclerotherapy

after patient is stabilized & intubated

---

Acute intoxication

Marijuana  appetite, dry mouth,

conjunctival injection,

tachycardia, slow reaction time, euphoria/dysphoria/paranoia, psychomotor impairment (days)

gynecomastia (chronic use)

phencyclidin e

P C P

violent behavior, impulsivity, hallucinations, amnesia, dissociation, vertical nystagmus, mydriasis, ataxia LSD visual hallucinations, mydriasis, euphoria/dysphoria/panic, perceptual intensification, tachycardia/palpitations/HTN, Cocaine euphoria, agitation,

formication (“cocaine bugs”)

chest pain, stroke, MI, seizures

tachycardia, HTN, mydriasis

Meth violent behavior, tooth decay

diaphoresis, tachycardia, HTN, choreiform movements, psychosis

Heroin

(opioid) euphoria, miosis,respiratory depression, depressed mental state,

constipation, hypotension, hypothermia, & bradycardia amphetamin

e agitation, paranoia, delirium, palpitations, tachycardia, HTN, diaphoresis, mydriasis, seizures, hyperthermia, intracerebral hemorrhage Alcohol slurred speech, unsteady gait,

disinhibition, nystagmus

 best predictor of opioid intoxication:  RR  Rx of severe psychomotor agitation a/w PCP

intoxication: benzo

---

cocaine withdrawal: dysphoria,  appetite,

hypersomnia, difficulty concentrating

opioid withdrawal: yawning, mydriasis,

lacrimation, rhinorrhea, diaphoresis, N/V, diarrhea, arthralgia, muscle spasms

---  recurrent fractures, hearing loss, opalescent teeth,

blue sclerae: osteogenesis imperfecta (AD)  normal intelligence, osteopenia

---  pain, followed by well-demarcated lesions & bullae

with skin necrosis: warfarin-induced necrosis  MC @ breasts, buttocks, thighs, abdomen  a/w protein C deficiency

Rx: vitamin K; discontinue warfarin if lesions progress, maintain anticoagulation with heparin ---  worsening renal function, HTN, & distal ischemia

following an invasive arterial procedure:

cholesterol embolization

 livedo reticularis on skin exam

---

plantar warts are due to HPV infection

 painful hyperkeratotic papules on the soles  common in young adults & immunocompromised ---  scaly, erythematous, ulcerated skin lesion that is

slow-growing, non-resolving with irregular growth on sun-exposed area: squamous cell ca

---  suspected appendicitis with delayed presentation

(> 5 days) after onset of symptoms: appendiceal

abscess (contained, perforated abscess = phlegmon)

Dx: CT scan

 if stable, Rx: hydration & IV ABX, bowel rest, elective appendectomy

---  anemia, constipation, weight loss: colon cancerfever, dysuria, flank pain: pyelonephritis ---

esophageal perforation MC occurs following

instrumentation of the esophagus o less commonly due to Boerhaave’s

 retrosternal pain & crepitus @ suprasternal notch due to pneumomediastinum

 DDx: Mallory-Weiss (self-limited hematemesis) ---

cat bites: prophylactic augmentin for 5 days

 Pasteurella multocida

---

fluoroquinolones is a/w tendon rupture in children

---  large thymic silhouette in the anterior mediastinum

is a normal finding on CXR in age < 3 yrs  triangular shape, “sail sign”, scalloped border  residual thymic tissue can undergo malignant

transformation into a thymoma  DDx: lymphoma

---

home O2 therapy & smoking cessation have

been shown to decrease mortality in COPD ---  hyperpigmentation of palmar creases, anorexia,

fatigue, GI complains, weight loss, hypotension: primary adrenal insufficiency (Addison’s)

 hyponatremia, hyperkalemia

hyponatremia is due to volume contraction

(aldosterone deficiency) & increased vasopressin (lack of cortisol suppression)

mild hyperchloremic acidosis

---  hypernatremia, hypokalemia: Cushing’s

---

Wolff-Parkinson-White: accessory pathway

that bypasses AV node; atrioventricular reentrant tachycardia (AVRT)

 persistent a-fib with rapid ventricular response can deteriorate to v-fib

Rx hemodynamically unstable: cardioversionRx hemodynamically stable: procainamide  AV nodal blockers (adenosine, β-blocker, CCB,

digoxin) can  accessory pathway conduction, thus worsen WPW

AV nodal reentry tachycardia (AVNRT) 2 separate conducting pathways (slow/fast) within AV node  sudden onset & termination, absent P, narrow QRS ---  abrupt onset of sharply-demarcated, edematous,

erythematous, tender skin lesion with raised border, & fever: erysipelas

 inflammation of superficial dermis  legs most common, or face  MCC: Group A strep

---

Todd’s paralysis: focal neurologic deficit

following a seizure

---  muscle weakness & dry mouth: Lambert-Eaton  a/w lung small cell carcinoma & Hodgkin’s  antibodies against presynaptic voltage-gated Ca++

channels in motor nerves

 muscle response to motor nerve stimulation increases with repetitive stimulation

diminished/absent DTRs

 Rx: plasmapheresis & immunosuppressants ---

Myasthenia crisis can be exacerbated by RTI

 diplopia, ptosis, proximal muscle weakness, & weakness of bulbar muscles & diaphragm can lead to respiratory distress

Rx MG with respiratory failure: intubation, then corticosteroids + IV Ig or plasmapheresis (preferred) ---

Myasthenic crisis: exacerbation of symptoms due

to undermedication of AChE-ases or infection  Rx: withhold AChE-ase inhibitors

(pyridostigmine), remove respiratory secretions

Cholinergic crisis: exacerbation of symptoms due

to overmedication with AChE-ases

Rx: atropine (anticholinergic, prevents A/E of AChE inhibitor therapy)

 Dx: edrophonium (Tensilon) test (short-acting AChE-inhibitor)

o myasthenic crisis  improves muscle strength o MG cholinergic crisis  worsens weakness ---  polyuria & polydipsia are classic for new-onset

Type I DM

 bimodal onset: age 4 – 6 yrs or early puberty

nocturnal enuresis can be a presenting

symptom in toddlers

 DDx: UTI, maturational delay of sphincter, psychological stress, nephrogenic DI

---

enuresis: urinary incontinence in age ≥ 5 yrs

primary enuresis: never achieved dryness

secondary enuresis: return of incontinence after

≥ 6 months of dryness

---  immune-mediated muscle inflammation:

polymyositis

 multicentric CNS inflammation & demyelination:

MS

---  infertility: inability to conceive for > 1 yr

infertility for women age > 35: > 6 months o MCC of fertility in the 4th decade who are

menstruating: age-related decreased

ovarian reserve

 Dx: early follicular phase FSH level, clomiphene challenge test, or inhibin-B level

 DDx: premature ovarian failure (amenorrhea & menopause before age 40)

---  rapid & massive increase in transaminases with

modest elevations in total bilirubin & ALP in the setting of hypotension (septic shock, heart failure):

ischemic hepatic injury

 DDx: alcoholic liver disease (AST rarely >300), acute hepatitis (significant hyperbilirubinemia), TB, sarcoidosis, acalculous cholecystitis, autoimmune hepatitis (elevated bilirubin) ---  if suspicion for subarachnoid hemorrhage is high,

but head CT is negative  lumbar puncture ---  sudden & unexpected travel, confusion about

personal identity, inability to remember the past:

dissociative fugue

 episodes of inability to recall important personal info; related to a traumatic or stressful event:

dissociative amnesia

 two or more distinct identities that alternatively assume control behavior: dissociative identity

disorder (multiple personality disorder)

 persistent/recurrent feelings of detachment from one’s own physical or mental processes, with intact sense of reality; significant impairment:

depersonalization disorder

 experiencing familiar persons & surroundings as if strange or unreal: derealization disorder ---  proximal DVT of lower extremities; MCC of PE o continue warfarin for 3 months for patients

with reversible risk factors

o continue warfarin for 6 – 12 months for

idiopathic DVT

---  glucose-6-phosphatase deficiency (Von-Gierkes’):

Type I glycogen storage disease  affects liver, kidneys, intestinal mucosa

 @ age 3 – 4 months, hypoglycemia, lactic acidosis, hyperuricemia, hyperlipidemia

 doll-like face (fat cheeks), thin extremities, protuberant abdomen, short stature

hypoglycemic seizures

---  Acid maltase deficiency (Pompe’s): Type II

glycogen storage disease

“floppy baby” with feeding difficulties, macroglossia, cardiomegaly, hepatomegaly ---  glycogen debranching enzyme deficiency: Type

III glycogen storage disease

elevated liver transaminases, fasting ketosis, normal blood lactate & uric acid levels

---  branching enzyme deficiency: Type IV glycogen

storage disease (amylopectinosis)

 age 18 months, hepatosplenomegaly, failure to thrive, progressive liver cirrhosis

---  new onset neurologic deficits (confusion),

occipital headaches, Hx of HTN & a-fib & vascular disease, on warfarin: strokeDx: non-contrast head CT

o hemorrhagic stroke  white hyperdense regions ---  all patients with B/L carpal tunnel syndrome

(CTS) should be screened with TSH

CTS 2/2 hypothyroidism is a/w deposition of mucopolysaccharide protein complexes within perineurium & endoneurium of median nerve;

commonly B/L, more severe

---  accumulation of fluid can cause CTS in pregosamyloid fibril deposition causes CTS in systemic

amyloidosis (ESRD, chronic hemodialysis)

 soft-tissue enlargement due to synovial edema & tendon hyperplasia causes CTS in acromegaly  inflammation of tendon & synovial sheaths cause

CTS in RA

---  severe hypovolemic hypernatremia Rx: isotonic

normal saline or lactated Ringer’s

 hypernatremia causes weakness, lethargy, irritability, altered mental status, seizures, muscle cramps & decreased DTRs

---  periodic abdo pain, multiple duodenal ulcers & a

jejunal ulcer: Zollinger-Ellison syndrome

steatorrhea due to pancreatic enzyme inactivation

---  pancreatic enzyme deficiency: chronic pancreatitisreduced bile salt absorption: ileal resection

---

hyperventilation decreases CO2 concentration

 promotes vasoconstriction  syncope

---

brief psychotic

disorder > 1 day, < 1 month; sudden onset, return to full function

Schizophreniform > 1 month, < 6 months;

functional decline not required

Schizophrenia at least 6 months;

functional decline required

Schizoaffective at least 2 week Hx of psychotic

Documento similar